Female Athlete Triad - Triad Components - Osteoporosis

Osteoporosis

Osteoporosis is defined by the National Institutes of Health as ‘‘a skeletal disorder characterized by compromised bone strength predisposing a person to an increased risk of fracture.’’ Low estrogen levels and poor nutrition, especially low calcium intake, can lead to osteoporosis, the third aspect of the triad. This condition can ruin a female athlete's career because it may lead to stress fractures and other injuries.

Immediate Effects of Low Bone Density. Patients with female athlete triad get osteoporosis due to hypoestrogenmia, or low estrogen levels which is part of amenorrhea. With estrogen deficiency, the osteoclasts live longer and are therefore able to resorb more bone. In response to the increased bone resorption, there is increased bone formation and a high-turnover state develops which leads to bone loss and perforation of the trabecular plates. As osteoclasts break down bone, patients see a loss of bone mineral density. Low bone mineral density renders bones more brittle and hence susceptible to fracture. Because athletes are active and their bones must endure mechanical stress, the likelihood of experiencing bone fracture is particularly high.

Additionally, because those suffering with female athlete triad are also restricting their diet, they may also not be consuming sufficient amounts vitamins and minerals which contribute to bone density; not getting enough calcium or vitamin D further exacerbates the problem of weak bones.

Long Term Effects of Low Bone Density. Bone mass is now thought to peak between the ages of 18-25. Thus, behaviors which result in low bone density in youth could be detrimental to an athlete’s bone health throughout her lifetime.

Read more about this topic:  Female Athlete Triad, Triad Components

Other articles related to "osteoporosis":

Osteoporosis - History
... at least to Astley Cooper, and the term "osteoporosis" and recognition of its pathological appearance is generally attributed to the French pathologist Jean Lobstein ... The American endocrinologist Fuller Albright linked osteoporosis with the postmenopausal state ... which revolutionized the treatment of osteoporosis, were discovered in the 1960s ...
Teriparatide - Uses
... for use in postmenopausal women with osteoporosis at a high risk for fracture or with a history of osteoporotic fracture, patients with multiple risk ... increase bone mass in men with primary or hypogonadal osteoporosis at high risk of fracture, patients with multiple risk factors for fracture, and for patients who have failed or are intolerant to other ... Teriparatide is indicated as well for the treatment of men and women with osteoporosis associated with sustained systemic glucocorticoid therapy ...
Other Lipid Hypotheses - Lipid Hypothesis of Osteoporosis
... The "lipid hypothesis of osteoporosis" postulates that lipids involved in causing heart disease also contribute to causing osteoporosis ... Osteoporosis is characterized by a decrease of bone marrow cells, or osteoblasts, and an increase of fat cells, or adipocytes ... Observations from this model suggest that LDL oxidation products can cause osteoporosis through changing the developmental fate of bone cells leading to a reduced number of ...
Senile Osteoporosis - Etiology
... considerations regarding senile osteoporosis are not very clear for physicians yet ... shown to play an important role in the pathogenetic process attached to senile osteoporosis as they enhance the action of the parathyroid hormone ... absorption of vitamin D concerning metabolism, are also factors that contributes to a diagnosis of osteoporosis type II ...
Steroid-induced Osteoporosis
... Steroid-induced osteoporosis (SIOP) is osteoporosis arising due to use of glucocorticoids - analogous to Cushing's syndrome and involving mainly the axial skeleton ... Osteoporosis may arise after taking steroids ...